Download or read book The Effect of Moderate Vs Heavy intensity Priming Exercise on Oxygen Uptake Kinetics During Constant Work Rate Cycling written by Anna M. Kinzel and published by . This book was released on 2018 with total page 120 pages. Available in PDF, EPUB and Kindle. Book excerpt: Background: Warming-up prior to exercise, sometimes referred to as priming exercise, is a common practice for most athletes. Heavy-intensity priming exercise (HIPE) has been shown to improve pulmonary oxygen uptake kinetics during a subsequent bout of heavy work compared to moderate-intensity priming exercise (MIPE). Less is understood about the effect of priming on regional muscle oxygen response (rMO2). Purpose: To determine if there is a significant difference between MIPE and HIPE on VO2 and rMO2 during constant work rate cycling (CWR)

Download or read book Oxygen Uptake Kinetics in Sport Exercise and Medicine written by Andrew M. Jones and published by Routledge. This book was released on 2013-07-04 with total page 547 pages. Available in PDF, EPUB and Kindle. Book excerpt: Despite its crucial importance, scientists interested in the limitations of human physical performance have only just started to give the field of oxygen uptake kinetics the attention it deserves. Understanding the principal determinant of the oxygen uptake kinetics is fundamental to improving human performance or the quality of life. This book provides a detailed overview of the current state of knowledge of this emerging field of study, and features: * an introduction to oxygen uptake kinetics and historical development of the discipline * measurement and analysis of oxygen uptake kinetics * control of and limitations to oxygen uptake kinetics * applications of oxygen uptake kinetics in a range of human populations. Oxygen Uptake Kinetics in Sport, Health and Medicine is richly illustrated and structured to enable easy access of information and represents an invaluable resource for students and researchers in exercise physiology, as well as for respiratory physiologists and pulmonary clinicians.

Download or read book Oxygen Uptake Kinetics During Heavy Cycling Versus Knee extension Exercise and Effect of Prior Heavy Exercise written by Sarah M. Cleland and published by . This book was released on 2008 with total page 168 pages. Available in PDF, EPUB and Kindle. Book excerpt:

Download or read book High Intensity Exercise in Hypoxia Beneficial Aspects and Potential Drawbacks written by Olivier Girard and published by Frontiers Media SA. This book was released on 2018-01-25 with total page 169 pages. Available in PDF, EPUB and Kindle. Book excerpt: In the past, ‘traditional’ moderate-intensity continuous training (60-75% peak heart rate) was the type of physical activity most frequently recommended for both athletes and clinical populations (cf. American College of Sports Medicine guidelines). However, growing evidence indicates that high-intensity interval training (80-100% peak heart rate) could actually be associated with larger cardiorespiratory fitness and metabolic function benefits and, thereby, physical performance gains for athletes. Similarly, recent data in obese and hypertensive individuals indicate that various mechanisms – further improvement in endothelial function, reductions in sympathetic neural activity, or in arterial stiffness – might be involved in the larger cardiovascular protective effects associated with training at high exercise intensities. Concerning hypoxic training, similar trends have been observed from ‘traditional’ prolonged altitude sojourns (‘Live High Train High’ or ‘Live High Train Low’), which result in increased hemoglobin mass and blood carrying capacity. Recent innovative ‘Live Low Train High’ methods (‘Resistance Training in Hypoxia’ or ‘Repeated Sprint Training in Hypoxia’) have resulted in peripheral adaptations, such as hypertrophy or delay in muscle fatigue. Other interventions inducing peripheral hypoxia, such as vascular occlusion during endurance/resistance training or remote ischemic preconditioning (i.e. succession of ischemia/reperfusion episodes), have been proposed as methods for improving subsequent exercise performance or altitude tolerance (e.g. reduced severity of acute-mountain sickness symptoms). Postulated mechanisms behind these metabolic, neuro-humoral, hemodynamics, and systemic adaptations include stimulation of nitric oxide synthase, increase in anti-oxidant enzymes, and down-regulation of pro-inflammatory cytokines, although the amount of evidence is not yet significant enough. Improved O2 delivery/utilization conferred by hypoxic training interventions might also be effective in preventing and treating cardiovascular diseases, as well as contributing to improve exercise tolerance and health status of patients. For example, in obese subjects, combining exercise with hypoxic exposure enhances the negative energy balance, which further reduces weight and improves cardio-metabolic health. In hypertensive patients, the larger lowering of blood pressure through the endothelial nitric oxide synthase pathway and the associated compensatory vasodilation is taken to reflect the superiority of exercising in hypoxia compared to normoxia. A hypoxic stimulus, in addition to exercise at high vs. moderate intensity, has the potential to further ameliorate various aspects of the vascular function, as observed in healthy populations. This may have clinical implications for the reduction of cardiovascular risks. Key open questions are therefore of interest for patients suffering from chronic vascular or cellular hypoxia (e.g. work-rest or ischemia/reperfusion intermittent pattern; exercise intensity; hypoxic severity and exposure duration; type of hypoxia (normobaric vs. hypobaric); health risks; magnitude and maintenance of the benefits). Outside any potential beneficial effects of exercising in O2-deprived environments, there may also be long-term adverse consequences of chronic intermittent severe hypoxia. Sleep apnea syndrome, for instance, leads to oxidative stress and the production of reactive oxygen species, and ultimately systemic inflammation. Postulated pathophysiological changes associated with intermittent hypoxic exposure include alteration in baroreflex activity, increase in pulmonary arterial pressure and hematocrit, changes in heart structure and function, and an alteration in endothelial-dependent vasodilation in cerebral and muscular arteries. There is a need to explore the combination of exercising in hypoxia and association of hypertension, developmental defects, neuro-pathological and neuro-cognitive deficits, enhanced susceptibility to oxidative injury, and possibly increased myocardial and cerebral infarction in individuals sensitive to hypoxic stress. The aim of this Research Topic is to shed more light on the transcriptional, vascular, hemodynamics, neuro-humoral, and systemic consequences of training at high intensities under various hypoxic conditions.

Download or read book The Effect of Muscle Mass During Priming Exercise on Pulmonary Oxygen Uptake and Cardiac Output Kinetics written by Ryan Seeto and published by . This book was released on 2012 with total page pages. Available in PDF, EPUB and Kindle. Book excerpt:

Download or read book Metabolic Responses to Supramaximal Exercise and Training written by Clare L. Weber and published by . This book was released on 2002 with total page pages. Available in PDF, EPUB and Kindle. Book excerpt: Abstract: The primary aim of this thesis was to investigate the gender-specific responses to supramaximal cycling and to examine the changes in anaerobic and aerobic metabolism that occur in response to high-intensity interval training (HIT). All subjects in the present experiments were untrained, healthy young adults aged between 18 and 35 yr. Cycle ergometry was used for all experimental test procedures and training programs. The accumulated oxygen (AO2) deficit was used to quantify the production of adenosine triphosphate (ATP) via anaerobic metabolism during supramaximal cycling. In addition, pulmonary oxygen uptake measured at the onset of exercise was described using mathematical modeling to determine the rate response of the aerobic energy system during exercise. The purpose of experiment one was to examine the test-retest reliability of the maximal accumulated oxygen deficit (MAOD) measured at 110% and 120% of peak oxygen uptake for cycling in seven untrained male and seven untrained female subjects. After one familiarization trial, all subjects performed two MAOD tests at a power output corresponding to 110% and two tests at 120% of peak oxygen uptake in random order. MAOD was calculated for each subject as the difference between the estimated AO2 demand and the AO2 uptake measured during the exercise bout. The meanplus or minusstandard error time to exhaustion (TE) for the group was not significantly different between trial one (226plus or minus13 s) and trial two (223plus or minus14 s) of the 110% test. Likewise, the difference in the TE between trial one (158plus or minus11 s) and trial two (159plus or minus10 s) was not significant for the 120% test. The intra-class correlation coefficients for the TE were 0.95 for the 110% test and 0.98 for the 120% test. The mean MAOD value obtained in trial one (2.62plus or minus0.17 L) was not significantly different from the mean value obtained in trial two (2.54plus or minus0.19 L) for the 110% test. Additionally, the mean values for the two trials did not differ significantly for MAOD (2.64plus or minus0.21 L for trial one and 2.63plus or minus0.19 L for trial two) in the 120% test. The intra-class correlation coefficients for MAOD were 0.95 for the 110% test and 0.97 for the 120% test. All intra-class correlation coefficients were significant at p less than 0.001. When conducted under standardized conditions, the determination of MAOD for cycling was highly repeatable at both 110% and 120% of peak oxygen uptake in untrained male and female subjects. The results observed in experiment one suggest that the MAOD may be used to compare the anaerobic capacity (AC) of men and women and to examine changes in the contribution of the anaerobic energy systems before and after training. Experiment two examined the gender-specific differences in MAOD before and after 4 and 8 wk of HIT. Untrained men (n=7) and women (n=7) cycled at 120% of pre-training peak oxygen uptake to exhaustion (MAOD test) pre-, mid-, and post-training. A post-training timed test was also completed at the MAOD test power output, but this test was stopped at the TE achieved during the pre-training MAOD test. The 14.3plus or minus5.2% increase in MAOD observed in males after 4 wk of training was not different from the 14.0plus or minus3.0% increase seen in females (p greater than 0.05). MAOD increased by a further 6.6plus or minus1.9% in males and this change was not different from the additional 5.1plus or minus2.3% increase observed in females after the final 4 wk of training. Peak oxygen uptake measured during incremental cycling increased significantly (p less than 0.01) in male but not in female subjects after 8 wk of training. Moreover, the AO2 uptake was higher in men during the post-training timed test compared to the pre-training MAOD test (p less than 0.01). In contrast, the AO2 uptake was unchanged from pre- to post-training in female subjects. The increase in MAOD with training was not different between men and women suggesting an enhanced ability to produce ATP anaerobically in both groups. However, the increase in peak oxygen uptake and AO2 uptake obtained in male subjects following training indicates improved oxidative metabolism in men but not in women. It was concluded that there are basic gender differences that may predispose males and females to specific metabolic adaptations following an 8-wk period of HIT. Increases in AO2 uptake during supramaximal cycling demonstrated in men after training led to the hypothesis that peak oxygen uptake kinetics are speeded in male subjects with short-term HIT. It was suggested that training does not improve peak oxygen uptake kinetics in women as no change in AO2 uptake was found after 8 wk of HIT in female subjects. The purpose of experiment three was to examine peak oxygen uptake kinetics before and after 8 wk of HIT in six men and six women during cycling at 50% (50% test) and 110% (110% test) of pre-training peak oxygen uptake. A single-term exponential equation was used to model the peak oxygen uptake response (after phase I) during the 50% and 110% tests pre- and post-training. In addition, phase II and III of the peak oxygen uptake response during the 110% tests were examined using a two-term equation. The end of the phase I peak oxygen uptake response was identified visually and omitted from the modeling process. The duration of phase I determined during all experimental tests was not different between men and women and did not change with training in either group. Before training, men obtained a phase II peak oxygen uptake time constant (t2) of 29.0plus or minus3.3 s during the 50% test which was not different to the t2 of 28.8plus or minus2.2 s attained by women. In addition, the t2 determined during the 50% test was unchanged after 8 wk of HIT in both groups. The peak oxygen uptake kinetics examined during the 110% tests before training were well described by a single-term model in all male and female subjects. The t2 determined before training for the 110% test was significantly faster in men than in women. Furthermore, peak oxygen uptake was unchanged in female subjects and the t2 remained unaltered with 8 wk HIT (pre 45.5plus or minus2.2; post 44.8plus or minus2.3 s). In contrast, male subjects achieved a significantly higher peak oxygen uptake after training and the t2 determined for men during the 110% test was faster after training (36.4plus or minus1.6 s) than before training (40.1plus or minus 1.9 s). Improved model fits were obtained with the two-term equation compared to the single-term equation in two of the six male subjects during the 110% test post-training. It was found that the onset of the peak oxygen uptake slow component occurred at a mean time of 63.5plus or minus2.5 s and the t2 was reduced to 18.4plus or minus1.7 s. Using a Wilcoxon Signed Ranks z-test, the t2 described by the single-term equation in the remaining four subjects was determined to be significantly faster after training than before training, thus confirming the results obtained from the original group (n=6) of male subjects. End exercise heart rate (HREE) values obtained during the 50% and 110% tests were not different between men and women. During the 50% test, HREE values were unchanged, whereas HREE was significantly decreased during the 110% test after training in both groups. These data show that HIT might improve oxidative metabolism in men but not in women as reflected by a greater peak oxygen uptake and faster peak oxygen uptake kinetics during supramaximal work rates. We further suggest that the faster peak oxygen uptake kinetics demonstrated in men after training are probably not due to an improvement in cardiac function. Finally, the augmentation of oxidative metabolism during exercise after HIT in men might be dependent on the intensity of the exercise bout at which the peak oxygen uptake response is examined. The findings presented in this thesis suggest that MAOD is a reliable measure in both male and female subjects and can be used to monitor changes in anaerobic ATP production during supramaximal cycling. Moreover, these data suggest that 4 and 8 wk of HIT produce similar changes in anaerobic ATP generation in men and women. Finally, 8 wk of HIT results in the increase of peak oxygen uptake and AO2 uptake as well as the speeding of peak oxygen uptake kinetics during supramaximal cycling in male subjects. There was no evidence to suggest that oxidative metabolism was improved in women after short-term HIT. -- In conclusion, improvement in supramaximal exercise performances should be examined specifically for changes in the anaerobic and aerobic contributions to energy production. In addition, it is suggested that gender should be of primary consideration when designing exercise-training programs where improvement in both anaerobic and aerobic metabolism is required.

Download or read book Oxford Textbook of Children s Sport and Exercise Medicine written by Neil Armstrong and published by Oxford University Press. This book was released on 2023 with total page 785 pages. Available in PDF, EPUB and Kindle. Book excerpt: The 4th edition of the Oxford Textbook of Children's Sport and Exercise Medicine is the definitive single-volume reference in the field presented in four sections Exercise Science; Exercise Medicine; Sport Science; and Sport Medicine.

Download or read book Effect of Systemic Acidosis on Oxygen Uptake Kinetics During High Intensity Cycling written by Karen Michal Wooten and published by . This book was released on 2006 with total page 64 pages. Available in PDF, EPUB and Kindle. Book excerpt:

Download or read book Science and Application of High Intensity Interval Training written by Laursen, Paul and published by Human Kinetics. This book was released on 2019 with total page 672 pages. Available in PDF, EPUB and Kindle. Book excerpt: The popularity of high-intensity interval training (HIIT), which consists primarily of repeated bursts of high-intensity exercise, continues to soar because its effectiveness and efficiency have been proven in use by both elite athletes and general fitness enthusiasts. Surprisingly, few resources have attempted to explain both the science behind the HIIT movement and its sport-specific application to athlete training. That’s why Science and Application of High-Intensity Interval Training is a must-have resource for sport coaches, strength and conditioning professionals, personal trainers, and exercise physiologists, as well as for researchers and sport scientists who study high-intensity interval training.

Download or read book ACSM s Metabolic Calculations Handbook written by Stephen Glass (Ph. D.) and published by Lippincott Williams & Wilkins. This book was released on 2007 with total page 132 pages. Available in PDF, EPUB and Kindle. Book excerpt: This handbook provides a step-by-step approach to using metabolic equations, from basic math principles to applying the equations to an exercise plan. Chapters focus separately on each equation, provide an easy-to-follow process of solving, and demonstrate the varied uses of the equation in clinical as well as fitness settings. Each chapter includes a set of problems that focus on real-world applications of the equation. Step-by-step problem solution explanations are provided at the end of each chapter. A comprehensive exam at the end of the book tests the reader's skill in using the equations.

Download or read book Effect of Pedal Rate During Time Trial on Oxygen Uptake Kinetics in a Subsequent Running Task written by Felix [Verfasser] Waldhuber and published by . This book was released on 2016 with total page pages. Available in PDF, EPUB and Kindle. Book excerpt: Der menschliche Körper befindet sich nur sehr selten in einem Zustand der Ruhe. Kommt es zu einer Veränderung der Belastung, z.B. durch einen sprunghaften Anstieg der Belastung beim Start eines sportlichen Wettkampfes, spielt die Kinetik der Sauerstoffaufnahme eine entscheidende Rolle für die dadurch erforderlichen Reaktionen des Organismus. Der Anstieg der Sauerstoffaufnahme infolge der Belastungssteigerung verläuft exponentiell und wird in drei Phasen unterteilt, welche durch die Zeitkonstante Tau () und die Amplitude näher beschrieben werden. Die Zeitkonstante ist in weiterer Folge bedeutend für die Größe des entstehenden Sauerstoffdefizits. Eine schnellere Kinetik der Sauerstoffaufnahme, ein kleineres Tau, resultiert in einem geringeren Sauerstoffdefizit gleichbedeutend mit einer geringeren Beteiligung anaerober Energiebereitstellung und später einsetzender Ermüdung (Jones & Poole, 2005b). Das Ziel der vorliegenden Arbeit bestand darin, festzustellen, ob die Trittfrequenz im Zeitfahren Auswirkungen auf die Sauerstoffaufnahmekinetik einer anschließenden Laufbelastung hat. Zehn männliche ausdauertrainierte Probanden (Mittelwert ± SD, Alter: 27.5 ± 4.8 Jahre, Körpergewicht: 76.2 ± 8.8 kg, V O2max: 62.1 ± 6.0 ml.kg-1.min-1) absolvierten drei Testungen unter Laborbedingungen. Nach einem Stufentest auf dem Laufband wurde an zwei weiteren Terminen jeweils eine kombinierte Belastung bestehend aus einem Zeitfahren auf dem Radergometer und einer anschließenden Laufbelastung. Beim Zeitfahren musste eine Arbeit von 360 Kilojoule (kJ) mit 60 und mit 90 Umdrehungen pro Minute (U/min) im linearen Modus des Lode-Ergometers so schnell wie möglich absolviert werden. Direkt nach Beendigung des Zeitfahrens wurden die Probanden gebeten, vom Ergometer zu steigen, in die Laufschuhe zu wechseln und die Laufbelastung am Laufband zu absolvieren. Diese umfasste sechs Minuten mit einer Geschwindigkeit entsprechend dem respiratorischen Kompensationspunkt (RCP), abgeleitet aus dem Stufentest. Aus der kontinuierlichen Messung der Sauerstoffaufnahme (Cortex MetaMax 3B) wurde in weiterer Folge ein monoexponentielles Modell zur Bestimmung der Zeitkonstante, der Amplitude und der Verzögerungszeit verwendet um die Kinetik der Sauerstoffaufnahme zu beschreiben. Der Effekt der Trittfrequenz wurde anhand eines T-Tests für gepaarte Stichproben analysiert. Es gab einen signifikanten Unterschied für die Dauer des Zeitfahrens (60 U/min: 1153 ± 141 s 90 U/min: 1201 ± 179 s t(9) = -2.877, p = 0.018). Es konnten keine signifikanten Effekte der Trittfrequenz auf die Zeitkonstante (60: 32.9 ± 6.5 s, 90: 33.8 ± 5.7 s t(9) = -1.12, p = 0.293), die Amplitude (Amplitude 60 = 4205 ± 492 ml, Amplitude 90 = 4232 ± 533 ml t(9) = 0.822, p = 0.433) und die Verzögerungszeit (TD 60 = 19.3 ± 2.4 s, TD 90 = 18.7 ± 3.0 s t(9) = 0.901, p = 0.391) festgestellt werden. Die Ergebnisse zeigen, dass die Trittfrequenz im Zeitfahren keine Auswirkung auf die Sauerstoffaufnahmekinetik einer anschließenden Laufbelastung hat.*****Oxygen uptake kinetics play a vital role whenever energetic demands of human muscles change due to a transition from rest to movement. The oxygen uptake rises in a near-exponential fashion. The response of oxygen uptake to a change in energetic demand is described by the time constant tau () and the amplitude. Faster oxygen uptake kinetics result in a lower oxygen deficit, reduced contribution of anaerobic energy supply and therefore reduced fatigue (Jones & Poole, 2005b). The aim of the present study was to analyze whether the pedal rate in a cycling time trial affects parameters of oxygen uptake kinetics in a subsequent running task under laboratory conditions. Ten male endurance trained subjects (mean ± SD, age: 27.5 ± 4.8 yr, body mass: 76.2 ± 8.8 kg, V O2max: 62.1 ± 6.0 ml.kg-1.min-1) performed three laboratory trials. After a graded exercise test (GXT) on a treadmill, two cycling time trials of 360 kJ followed by a 6-min running task were completed on separate

Download or read book Energetics of Muscular Exercise written by Guido Ferretti and published by Springer. This book was released on 2015-03-25 with total page 196 pages. Available in PDF, EPUB and Kindle. Book excerpt: This book discusses the maximal power and capacity of the three major biochemical pathways - aerobic (oxygen consumption), anaerobic lactic (muscle lactate accumulation in absence of oxygen consumption), and anaerobic alactic (phosphocreatine hydrolysis) metabolism - as well as the factors that limit them. It also discusses the metabolic and cardio-pulmonary mechanisms of the dynamic response to exercise. The way and extent to which the power and capacity of the three major energy metabolisms are affected under a number of different conditions, such as training, hypoxia and microgravity, are also described.

Download or read book The Effect of Age on the Relationship Amongst Oxygen Uptake Leg Blood Flow and Muscle Deoxygenation Kinetics During Moderate intensity Exercise written by Gregory R. duManoir and published by . This book was released on 2010 with total page 374 pages. Available in PDF, EPUB and Kindle. Book excerpt:

Download or read book Exercise Cognition Interaction written by Terry McMorris and published by Academic Press. This book was released on 2015-11-06 with total page 508 pages. Available in PDF, EPUB and Kindle. Book excerpt: Exercise-Cognition Interaction: Neuroscience Perspectives is the only book on the market that examines the neuroscientific correlation between exercise and cognitive functioning. The upsurge in research in recent years has confirmed that cognitive-psychology theory cannot account for the effects of exercise on cognition, and both acute and chronic exercise effect neurochemical and psychophysiological changes in the brain that, in turn, affect cognitive functioning. This book provides an overview of the research into these effects, from theoretical research through current studies that emphasize neuroscientific theories and rationales. It addition, users will find a thorough examination of the effects of exercise interventions on cognitive functioning in special populations, including the elderly, children, and those suffering from a variety of diseases, including schizophrenia, diabetes, and an array of neurological disorders. With contributions from leading researchers in the field, this book will be the go-to resource for neuroscientists, psychologists, medical professionals, and other researchers who need an understanding of the role exercise plays in cognitive functioning. Provides a comprehensive account of how exercise affects brain functioning, which in turn affects cognition Covers both theory and empirical research Presents a thorough examination of the effects of exercise interventions on cognitive functioning in special populations, including the elderly, children, and those suffering from a variety of diseases Examines neurochemical, psychophysiological, and genetic factors Covers acute and chronic exercise

Download or read book Monitoring Metabolic Status written by Institute of Medicine and published by National Academies Press. This book was released on 2004-08-29 with total page 469 pages. Available in PDF, EPUB and Kindle. Book excerpt: The U.S. military's concerns about the individual combat service member's ability to avoid performance degradation, in conjunction with the need to maintain both mental and physical capabilities in highly stressful situations, have led to and interest in developing methods by which commanders can monitor the status of the combat service members in the field. This report examines appropriate biological markers, monitoring technologies currently available and in need of development, and appropriate algorithms to interpret the data obtained in order to provide information for command decisions relative to the physiological "readiness" of each combat service member. More specifically, this report also provides responses to questions posed by the military relative to monitoring the metabolic regulation during prolonged, exhaustive efforts, where nutrition/hydration and repair mechanisms may be mismatched to intakes and rest, or where specific metabolic derangements are present.

Download or read book Principles of Exercise Testing and Interpretation written by Karlman Wasserman and published by Lww. This book was released on 2015-04-27 with total page 588 pages. Available in PDF, EPUB and Kindle. Book excerpt: "In this fifth edition of Principles of Exercise Testing and Interpretation, as in earlier editions, we attempt to develop conceptual advances in the physiology and pathophysiology of exercise, particularly as related to the practice of medicine. The underlying theme of the book continues to be the recognition that the most important requirement for exercise performance is transport of oxygen to support the bioenergetic processes in the muscle cells (including, of course, the heart) and elimination of the carbon dioxide formed as a byproduct of exercise metabolism. Thus, appropriate cardiovascular and ven- tilatory responses are required to match those of muscle respiration in meeting the energy demands of exercise. As depicted by the logo on the book cover, normal exercise performance requires an efficient coupling of external to internal (cellular) respiration. Appropriate treatment of exercise intolerance requires that patients' symptoms be thought of in terms of a gas exchange defect between the cell and the environment. The defect may be in the lungs, heart, peripheral or pulmonary circulations, the muscles themselves, or there may be a combination of defects. Thus, we describe the pathophysiology in gas transport and exchange that affect any site in the cardio- respiratory coupling between the lungs and the muscles. We illustrate how cardiopulmonary exercise testing can provide the means for a critical evaluation by the clinician-scientist of the functional competency of each component in the coupling of cellular to external respiration, including the cardiovascular system. To achieve this, clinical cases are used to illustrate the wide spectrum of pathophysiology capable of causing exercise intolerance"--Provided by publisher.

Download or read book Handbook of Sports Medicine and Science written by Jonathan C. Reeser and published by John Wiley & Sons. This book was released on 2008-04-15 with total page 245 pages. Available in PDF, EPUB and Kindle. Book excerpt: This addition to the Handbook series is presented in five sections. The first sections covers basic and applied science, including biomechanics, the physiologic demands of volleyball, conditioning and nutrition. The second section looks at the role of the medical professional in volleyball, covering team physicians, pre-participation examination, medical equipment at courtside and emergency planning. The third section looks at injuries - including prevention, epidemiology, upper and lower limb injuries and rehabilitation. The next section looks at those volleyball players who require special consideration: the young, the disabled, and the elite, as well as gender issues. Finally, section five looks at performance enhancement.